CCRN (Adult) guideCardiovascular and respiratory (25%)
CCRN Cardiovascular Review (13%): Dysrhythmias, ACS, Tamponade, and Hypertensive Crisis
What the 13% cardiovascular category of the CCRN (Adult) test plan covers, with a stable-or-unstable rhythm table from the 2025 AHA guidelines, a tamponade vignette, and the first-hour blood pressure arithmetic for a hypertensive emergency.
Short answer
Cardiovascular is 13% of the CCRN (Adult) test plan for exams taken on and after November 12, 2025, and AACN lists 16 cardiovascular patient problems under it. The decisions that keep coming back are whether a tachycardia or bradycardia is actually causing instability, which treatments the 2025 AHA guidelines label harmful, how cardiac tamponade looks at the bedside, and how far to lower blood pressure in the first hour of a hypertensive emergency. Under the 2025 AHA/ACC blood pressure guideline, a hypertensive emergency without a compelling condition such as aortic dissection calls for a systolic drop of no more than 25% in the first hour.
What the cardiovascular category covers on the CCRN exam
Cardiovascular is 13% of the Adult CCRN Test Plan that applies to exams taken on and after November 12, 2025. AACN publishes percentages, not item counts, so any count is an estimate: 13% of the 125 scored items is about 16 questions (Passy estimate). The category was 17% under the 2020 test plan, and the 2025 list places acute pulmonary edema under Respiratory rather than here.
The test plan pairs the 16 listed problems with "testable nursing actions." For this category they are interpreting and responding to cardiac rhythms, monitoring hemodynamic status, recognizing early signs of falling cardiac output, and caring for patients who need catheterization, endovascular procedures, mechanical circulatory support, invasive or noninvasive hemodynamic monitoring, pericardiocentesis or temporary pacing.
| Listed problems (paraphrased from the test plan) | Where to study them |
|---|---|
| Dysrhythmias, and conduction defects such as a prolonged QT interval or Wolff-Parkinson-White | The stable-or-unstable section below |
| Acute coronary syndrome | The ACS section below |
| Cardiac tamponade, cardiac surgery, cardiac trauma | The tamponade vignette below, plus the CCRN hemodynamics guide for the pressure pattern |
| Hypertensive crisis, and aortic aneurysm, dissection or rupture | The first-hour blood pressure section below |
| Cardiogenic shock, heart failure, cardiomyopathy | The sepsis and shock guide, which compares shock states, and the hemodynamics guide |
| Cardiac infection and inflammation, structural and valvular disease, vascular disorders, catheterization, vascular interventions | A critical care text from AACN's Adult bibliography, such as AACN Essentials of Critical Care Nursing (5th ed., 2023) |
A note on editions. AACN validates each exam item against at least one reference on its bibliography, and the Adult bibliography still lists the 2020 AHA Guidelines for CPR and ECC. This page teaches from the 2025 AHA guidelines. If your review book and this page disagree on a rhythm point, check which edition each one follows.
Stable or unstable: the first question in a CCRN rhythm item
Decide whether the rhythm is causing instability before you name it. Part 9 of the 2025 AHA Guidelines (Adult Advanced Life Support) lists hypotension, acutely altered mental status, signs of shock, chest pain and acute heart failure as signs of instability with a narrow-complex tachycardia (NCT). For a wide-complex tachycardia (WCT), its examples are a systolic pressure below 80 mm Hg or altered mentation. There is a second condition: the tachycardia must plausibly be the cause of the instability, not a response to another illness such as sepsis.
| Situation | What the 2025 AHA guideline recommends (class) | What it labels harmful |
|---|---|---|
| Regular NCT, stable | Vagal maneuvers and adenosine (1). IV diltiazem or verapamil can be effective (2a). Synchronized cardioversion if those fail or are contraindicated (1) | Nothing listed for this situation |
| NCT, unstable | Synchronized cardioversion (1) | Nothing listed for this situation |
| Monomorphic WCT with a pulse, unstable | Synchronized cardioversion (1) | Adenosine in unstable, irregularly irregular or polymorphic WCT. Verapamil and diltiazem in any WCT |
| Monomorphic WCT with a pulse, stable | Adenosine may be considered for a regular monomorphic WCT of unclear cause (2b). IV amiodarone, procainamide or sotalol may be considered (2b). Synchronized cardioversion if drugs fail or are contraindicated (1) | Verapamil and diltiazem |
| Sustained polymorphic VT | Immediate unsynchronized shock (1). Magnesium may be considered for recurrences with a long QT, that is, torsades de pointes (2b) | Routine magnesium with a normal QT is rated "no benefit" |
| Bradycardia with symptoms | Look for and treat reversible causes (1). Atropine if there is hemodynamic compromise (2a). If atropine fails, an epinephrine or dopamine infusion or transcutaneous pacing (2b) while transvenous pacing is prepared | Nothing listed for this situation |
For the exam, the nurse's part is recognition and readiness: identify the instability, call for help, and have the defibrillator ready in synchronized mode when cardioversion is the next step. The table gives no doses on purpose. Doses belong to your unit's protocol and the provider's order.
Two patients, one alarm (fictional). In bed 4, a 58-year-old woman two days after a bowel resection develops a sudden, regular narrow tachycardia at about 180/min. Her blood pressure is 118/74 mm Hg, she is alert, and she reports palpitations without chest pain. None of the AHA instability signs is present, so she is stable: vagal maneuvers and adenosine are the first options, and cardioversion waits until they fail.
In bed 9, a 66-year-old man with pneumonia has a temperature of 39.4 °C, a sinus tachycardia of 138/min and a blood pressure of 92/50 mm Hg. He is hypotensive, but the rhythm is a response to the infection, not its cause. Cardioversion would treat nothing here. The work is sepsis care, which the CCRN sepsis and shock guide lays out.
The free CCRN set includes a third version of this decision: a post-operative patient whose sudden narrow-complex tachycardia comes with low pressure and new confusion. When the table makes sense, test the unstable narrow-complex tachycardia decision in the free CCRN set and read why each other option falls short.
Acute coronary syndrome: what to recognize first
The MSD Manual's overview of acute coronary syndromes (reviewed July 2026), which cites the 2025 ACC/AHA ACS guideline, separates the three syndromes by symptoms, ECG findings and cardiac troponin:
- Unstable angina: symptoms of myocardial ischemia without evidence of acute myocardial infarction on the clinical assessment and serial troponin testing.
- NSTEMI: acute myocardial injury, shown by a rise or fall in troponin with at least one value above the assay's 99th-percentile upper reference limit, together with evidence of ischemia, without persistent diagnostic ST elevation. An elevated troponin alone is not enough.
- Suspected STEMI: an ischemic presentation with diagnostic ST elevation or a recognized occlusion-equivalent ECG pattern requires urgent reperfusion assessment. Do not wait for troponin confirmation or use the response to nitroglycerin to decide whether the ECG is an emergency.
Two time targets from the same source are worth knowing. The ECG should be done as soon as possible, within 10 minutes of presentation to medical care. For STEMI, emergency PCI is the choice when door-to-balloon time is under 90 minutes, or 120 minutes when the patient is transferred between hospitals. At the bedside that means getting the 12-lead fast, comparing it with any earlier tracing, and watching the ST segments continuously. AACN keeps a Practice Alert on this, "Ensuring Accurate ST-Segment Monitoring."
Cardiac tamponade after heart surgery: a worked vignette
The MSD Manual's pericarditis chapter (reviewed April 2026) describes tamponade findings that resemble cardiogenic shock (low cardiac output, low arterial pressure, tachycardia, dyspnea), and often includes distended neck veins. Venous distension is not unique to tamponade and can also occur with right-sided or biventricular pump failure. Severe tamponade is nearly always accompanied by pulsus paradoxus, an inspiratory fall in systolic pressure of more than 10 mm Hg, and the heart sounds are muffled unless the effusion is small. On a pulmonary artery catheter, diastolic pressures are elevated and roughly equal in all chambers and in the pulmonary artery.
A fictional patient. Six hours after mitral valve repair, a 71-year-old man's heart rate climbs from 92 to 126/min over an hour. His blood pressure falls from 108/64 to 84/62 mm Hg, his CVP rises from 8 to 16 mm Hg, and the pulmonary artery diastolic and wedge pressures now sit within a couple of points of the CVP. His neck veins are distended and his heart sounds are harder to hear than at the start of the shift.
Read the pattern, not one number:
- Filling pressures are rising while pressure and output fall. That combination points to a heart that cannot fill, not to fluid overload. The MSD Manual's critical care monitoring chapter notes that tamponade lowers cardiac output by reducing venous return and preload, so a diuretic or vasodilator aimed at the CVP number would work against him.
- The diastolic pressures are converging. Near-equal pressures across the chambers are the catheter's tamponade signature.
- Small volumes matter after surgery. MSD notes that rapid accumulation of as little as about 150 mL can cause tamponade. It also warns that a loculated effusion or a localized clot may compress only some chambers, so the classic signs can be missing.
- It is a clinical diagnosis. When tamponade is suspected, echocardiography is done unless even a brief delay could be life threatening, and then the fluid is drained immediately.
The nursing priority is to notify the surgeon now, stay with the patient, and prepare for emergency decompression as the team directs.
Hypertensive emergency: the first-hour arithmetic
Section 6.2 of the 2025 AHA/ACC high blood pressure guideline defines a hypertensive emergency as blood pressure above 180/120 mm Hg with evidence of acute target-organ damage, and recommends ICU admission (class 1). How far to lower the pressure in the first hour depends on whether a compelling condition is present.
| Situation (2025 AHA/ACC, section 6.2) | Goal | Class |
|---|---|---|
| Hypertensive emergency with a compelling condition, such as acute aortic syndrome | Systolic below 140 mm Hg in the first hour for most conditions | 1 |
| Acute aortic dissection | Systolic below 120 mm Hg in the first hour | 1 |
| Hypertensive emergency without a compelling condition | Lower systolic by no more than 25% in the first hour, then, if stable, to below 160/100 within the next 2 to 6 hours, then cautiously to 130–140 systolic over 24 to 48 hours | 1 |
| Severe hypertension (above 180/120) in a patient hospitalized for a noncardiac condition, with no acute target-organ damage | Extra intermittent IV or oral doses to lower the pressure acutely are not recommended | 3: Harm |
The guideline's reason for going slowly is autoregulation: in long-standing hypertension, a rapid return to normal can leave vital organs underperfused. Its supportive text adds that continuous infusions of short-acting, titratable agents are often preferable in the ICU. Stroke and pregnancy follow separate sections, and the CCRN neuro review covers the 2026 stroke targets.
Worked example (fictional). A 63-year-old arrives at 232/128 mm Hg with new acute kidney injury. Imaging shows no aortic dissection, there is no stroke, and she is not pregnant, so no compelling condition applies.
- 25% of 232 is 58.
- 232 − 58 = 174, so the first-hour goal is a systolic pressure no lower than about 174 mm Hg.
- If she stays stable, the next step is below 160/100 within 2 to 6 hours, then 130–140 systolic over the following 24 to 48 hours.
Had the scan shown an aortic dissection, the same reading would call for a systolic below 120 mm Hg within the hour. The patient's diagnosis, not the height of the number, sets the target. To check the rule on a new set of numbers, try the first-hour blood pressure goal item in the mixed CCRN set. Then see how this category fits with the others in the CCRN (Adult) test plan guide.
Trust the source
Official sources
Exam policies can change. Use these primary sources for the most current details.
- Adult CCRN Test Plan (exams on and after November 12, 2025)AACN Certification Corporation↗
- Adult CCRN Exam BibliographyAACN Certification Corporation↗
- Part 9: Adult Advanced Life Support. 2025 AHA Guidelines for CPR and ECC (Circulation 2025;152(suppl 2):S538–S577)American Heart Association↗
- 2025 AHA/ACC Guideline for the Management of High Blood Pressure in Adults (Hypertension 2025;82:e212–e316), section 6.2American Heart Association / American College of Cardiology↗
- Pericarditis, including cardiac tamponade (MSD Manual Professional Edition, reviewed April 2026)Merck & Co. (MSD Manual)↗
- Monitoring and Testing the Critical Care Patient (MSD Manual Professional Edition, reviewed October 2025)Merck & Co. (MSD Manual)↗
- Overview of Acute Coronary Syndromes (MSD Manual Professional Edition, reviewed July 2026)Merck & Co. (MSD Manual)↗
- Practice AlertsAmerican Association of Critical-Care Nurses↗
- Fourth Universal Definition of Myocardial Infarction (2018): acute myocardial injury and ischemia criteriaESC / ACC / AHA / WHF↗