CCRN (Adult) guideCardiovascular and respiratory (25%)
CCRN Hemodynamics: Pressures, Waveforms, and What Each Number Tells You
Normal PA catheter pressures from a named reference, how the waveform changes as the catheter advances, wedge safety, why CVP and wedge do not predict fluid response, and the pressure patterns that point to each shock state and to tamponade.
Short answer
Hemodynamic monitoring is a listed testable nursing action on the CCRN (Adult) test plan, so expect stems that hand you CVP, PA, wedge, cardiac output or SvO2 values and ask what they mean for the patient. Learn one set of normal ranges, read trends rather than single values, and know the patterns: low filling pressures with hypovolemia, a high wedge with left-sided heart failure, vasodilation in distributive shock, and elevated, nearly equal diastolic pressures in cardiac tamponade. AACN's PA/CVP practice alert adds that CVP and wedge pressure do not reliably predict who will respond to a fluid bolus.
Where hemodynamics sits on the CCRN (Adult) test plan
The Adult CCRN test plan for exams taken on and after November 12, 2025, puts Cardiovascular at 13% of the exam. Its list of testable nursing actions names invasive and non-invasive hemodynamic monitoring and mechanical circulatory support devices. Pressures also show up outside the Cardiovascular category: the Multisystem category (16%) lists shock states and sepsis, and those stems often come with a CVP or a cardiac output.
The skill the exam rewards is interpretation. You are given numbers and a patient, and you have to say which problem the numbers point to and what the nurse does next. That starts with knowing what normal looks like.
Normal pressures to anchor on
The values below come from the MSD Manual's critical care monitoring chapter (full review October 2025). References print slightly different ranges, so learn one set well and expect small differences elsewhere.
| Measurement | Normal range in the MSD Manual |
|---|---|
| Right atrial pressure (CVP) | 0 to 8 mm Hg |
| Right ventricle, peak systolic / end-diastolic | 15 to 30 / 0 to 8 mm Hg |
| Pulmonary artery, peak systolic / end-diastolic | 15 to 30 / 4 to 14 mm Hg |
| Pulmonary artery mean | 9 to 16 mm Hg |
| Pulmonary artery occlusion (wedge) pressure, mean | 2 to 12 mm Hg |
| Cardiac index | 3.5 ± 0.7 L/min/m² |
| Systemic vascular resistance | 1130 ± 178 dynes·s·cm⁻⁵ |
| Mixed venous oxygen saturation (SvO2) | 65% to 75% |
The same chapter makes a point that matters for exam stems: a changing trend usually means more than one abnormal value. A CVP that climbs from 8 to 16 over an hour tells you more than a single reading of 12.
How the PA catheter waveform changes as it advances
Knowing the insertion sequence helps you recognize where the catheter tip is from the trace alone. MSD describes it this way, in summary:
- Right atrium: a low pressure, close to the CVP.
- Right ventricle: the systolic pressure jumps suddenly to about 30 mm Hg, while the diastolic pressure stays close to the right atrial value.
- Pulmonary artery: the systolic pressure stays about the same, but the diastolic pressure rises above the right ventricular end-diastolic pressure, so the pulse pressure narrows.
- Wedge: the balloon lodges in a distal branch, and the trace now reflects back pressure from the pulmonary veins.
Once the catheter sits in the pulmonary artery, the balloon is deflated and a chest radiograph confirms placement. On the exam, the step that changes is the clue: a systolic jump marks entry into the ventricle, and a diastolic rise marks entry into the pulmonary artery.
Wedge safety and why some teams trend the PA diastolic
With the balloon inflated, the wedge pressure approximates left atrial pressure and, through it, left ventricular end-diastolic pressure. Two safety facts from the MSD chapter are worth memorizing:
- Use the minimum inflation needed for a valid wedge reading and deflate promptly according to the catheter instructions and unit protocol. MSD gives 30 seconds as an upper limit, not a recommended hold time, because a persistently wedged or overinflated balloon can cause pulmonary infarction.
- Pulmonary artery rupture is estimated at 0.03% to 0.2% of insertions, is often fatal, and happens at the moment of wedging. For that reason, many physicians prefer to follow the PA diastolic pressure as a stand-in for the wedge.
The two numbers usually track together, but the PA diastolic can run higher than the wedge when pulmonary vascular resistance is raised by lung disease. Wedge readings also misrepresent left ventricular filling in mitral stenosis or regurgitation, at PEEP above 10 cm H2O, and when left ventricular compliance changes.
CVP and wedge do not tell you who will respond to fluid
AACN's Practice Alert "PA/CVP Monitoring in Adults" (published August 1, 2016, last reviewed September 16, 2026) states in its public summary that PAOP and CVP are not accurate for identifying which patients will increase their stroke volume after a fluid bolus. It points instead to functional hemodynamic indicators, and it notes that adding CVP monitoring to guided therapy has not improved outcomes in severe sepsis and septic shock.
For the exam, this means a single static value does not answer the question "will a bolus help?" The better answer is usually the one that tests the response (a measured change in stroke volume or cardiac output after a challenge) or treats the cause the stem describes. The sepsis and shock guide covers the 2026 Surviving Sepsis approach to fluids and vasopressors.
Pressure patterns that point to each shock state
The table pairs each mechanism from the MSD Manual's shock chapter (full review June 2026) with what the MSD sources say the pressures show. Where the sources describe a finding in words rather than a number, the table does too.
| Shock state | Mechanism | What the numbers tend to show |
|---|---|---|
| Hypovolemic | Critical loss of intravascular volume; less venous return, smaller stroke volume | Low filling pressures: MSD says a CVP below 5 mm Hg or a wedge below 8 mm Hg may indicate hypovolemia. Cardiac output falls |
| Cardiogenic | Reduced cardiac output from a primary cardiac disorder (ischemia, dysrhythmia, acute valve or septal failure) | Low cardiac output. The wedge is elevated in left-sided heart failure |
| Distributive (septic, anaphylactic, neurogenic) | Arterial or venous vasodilation, so the vascular space is too large for the volume | Vasodilation lowers systemic vascular resistance. Skin may be warm or flushed, especially in sepsis, and hypoperfusion can occur even with a normal cardiac output |
| Obstructive | Mechanical interference with filling or emptying (tamponade, tension pneumothorax, pulmonary embolism) | Depends on the cause. In tamponade, diastolic pressures rise and equalize. In pulmonary embolism, bedside echo may show a dilated right ventricle |
SvO2 adds a second layer. A low value can reflect reduced oxygen delivery or increased demand; interpret it with hemoglobin, arterial oxygenation, cardiac output, and the clinical picture. If SaO2 is 97% and SvO2 is 52%, the saturation difference is 45 percentage points. That is not itself an oxygen-extraction percentage: ignoring dissolved oxygen and assuming comparable hemoglobin, the extraction ratio is approximately (97 − 52) / 97 = 46%. Trends and possible measurement limitations matter more than that calculation alone.
The tamponade pattern after cardiac surgery
Tamponade is the classic hemodynamics item, because the numbers carry the diagnosis. The MSD pericarditis chapter (full review April 2026) describes it this way:
- Findings: low cardiac output, hypotension, tachycardia, and markedly distended neck veins. Heart sounds are muffled unless the effusion is small, and severe tamponade is nearly always accompanied by an inspiratory drop in systolic pressure of more than 10 mm Hg (pulsus paradoxus).
- On a PA catheter: diastolic pressures are elevated, at roughly 10 to 30 mm Hg, and equal in all cardiac chambers and the pulmonary artery.
- Small volumes can be enough: if blood collects quickly, as little as about 150 mL can cause tamponade, because the pericardium cannot stretch fast enough.
- It is a clinical diagnosis: echocardiography is done unless even a brief delay could be life threatening, and drainage follows immediately.
Here is how that reads at the bedside, in an original example. Six hours after valve surgery, a patient's heart rate rises from 96 to 124/min and the arterial pressure drifts from 104/62 to 82/58 mm Hg. Over the same hour the CVP climbs from 9 to 17 mm Hg, the PA diastolic reads 18, and a wedge check reads 17. Three filling pressures that started at different levels have converged at a high value while output falls. That convergence, with falling pressure and rising heart rate, should make you think of tamponade before heart failure or fluid overload.
The nursing priorities follow from the physiology. Notify the surgeon immediately and prepare for emergency decompression. A diuretic for the "high CVP" or a vasodilator to "unload the heart" treats the wrong problem, because the heart is being squeezed, not overfilled. The free 20-question CCRN (Adult) set includes a post-cardiac-surgery item with near-equal filling pressures, so once this pattern makes sense, check your tamponade call against the equal-pressure scenario and read why each other option fails.
Putting the numbers together on exam day
When a stem gives you hemodynamic values, work in this order:
- Compare each value with normal and note the direction of change since the last reading.
- Ask which mechanism fits: too little volume, a failing pump, a vascular space that is too large, or an obstruction.
- Check the pattern against the history. A post-cardiac-surgery patient with converging pressures is a different problem from a septic patient with warm skin and a low SVR.
- Choose the action that treats the mechanism, not the one that normalizes a single number.
The cardiovascular review covers the dysrhythmia and hypertensive crisis items that often sit next to hemodynamics questions in a study session.
Trust the source
Official sources
Exam policies can change. Use these primary sources for the most current details.
- Adult CCRN Test Plan (exams taken on and after November 12, 2025)AACN Certification Corporation↗
- CCRN Exam Handbook – Direct Care (July 2026)AACN Certification Corporation↗
- Monitoring and Testing the Critical Care Patient (full review October 2025)MSD Manual Professional Version↗
- Shock (full review June 2026)MSD Manual Professional Version↗
- Pericarditis, including cardiac tamponade (full review April 2026)MSD Manual Professional Version↗
- Practice Alert: PA/CVP Monitoring in Adults (published 8/1/2016, last reviewed 9/16/2026)American Association of Critical-Care Nurses↗